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4E, F), which were localized to autophagosomal and lysosomal membranes, respectively, and were used to indicate autophagosome–lysosome fusion [38]

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As both neuroinflammation and oxidative stress or metabolic dysregulation are prevalent across the epilepsies, which are both argued to be causal and consequential to the disease, the direct interactions between neuroinflammation and oxidative stress are key components of disease pathology which deserve further evaluation